‘Leukoplakia’

Oral Hairy Leukoplakia (LVO) part 2

Monday, March 8th, 2010

dentist LVOHow is the LVO?

LVO usually does not require treatment or cause serious symptoms. However, treatment may be an option for those concerned with the appearance of the LVO in the language, or who have disseminated lesions and experience discomfort or taste changes due to the plates.

The usual method for treating viral LVO is taken orally (by mouth). Usually, these medicines are taken for one to two weeks or until the plates of the LVO disappear.
Other options include tretinoin (Retin A) and podophyllin resin, two drugs that can be applied directly to the plates of LVO. Usually tretinoin is applied two or three times a day until the plates are gone. Podophyllin it operates a health care provider once or twice during a period of two to three weeks. Other options, especially if the plates are small, is that a healthcare provider applies liquid nitrogen (cryotherapy) to the affected area, or surgery to remove the plates.

LVO oral leukoplakiaCan you prevent the LVO?

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Oral Hairy Leukoplakia (LVO) part 1

Friday, March 5th, 2010

oral hairy leukoplakiaWhat is it?

Oral hairy leukoplakia (LVO) is one or more white patches that can appear in the mouth. Normally, these plaques appear along the border of the tongue, but sometimes develop on the top or bottom of the tongue or along the inside of the cheek. If you look closely, these plaques can appear hairy or contain a number of small folds or bumps.

The LVO may look like thrush (oral candidiasis), another common problem characterized by white patches that develops in the mouths of people with HIV. However, if canker scraped gently using a toothbrush, usually are removed, the LVO not.

The LVO is one of the first opportunistic infections that occur in people with HIV. May occur with any T-cell count While there are people with HIV with more than 500 T cells that developed LVO, is more common in those with T-cell counts under 200. It is noteworthy that the LVO may affect those with healthy immune systems, even those who are not infected with HIV.

It is considered a benign disease, since it rarely causes serious physical problems and cause no serious complications.

The Epstein-Barr virus (EBV) is causing the LVO. Most people worldwide are infected with EBV. But only some, including those with a weakened immune system, develop the disease.

Over 25% of HIV positive people LVO develops sometime during the course of infection. It is more common among HIV positive men and smokers.

oral hairy leukoplakiaWhat are the symptoms of LVO?

The classic symptom of the LVO are looking fluffy white patches (hairy) and bumps that develop on the edge of the tongue. Plates may also appear at the top or bottom of the tongue, or along the inside of the cheek. It is also possible that these plates do not exhibit villi and contain no visible bumps or creases.

In general, LVO causes no other symptoms. Therefore, many people may not know they have LVO unless you consider the tongue or inside the mouth to find the plates, which usually do not cause discomfort or affect the taste of food or liquids. In some cases, LVO may cause mild pain, taste changes and sharpen sensitivity to food temperatures.

How is it diagnosed the LVO?

Often, health professionals can diagnose by simply looking LVO white plates. To find out if LVO plates or thrush (candidiasis) may be a simple test of scaling. With the help of a trowel or brush teeth gently scraping the plate. If the board is clear with white scaling, it is probably thrush, not LVO.

To be sure if the white patch is LVO, your health care provider can send a sample to a lab for analysis. The lab will look for the Epstein-Barr virus (EBV) to confirm the diagnosis of LVO.

Incoming search terms for the article:

inside mouth bottom of cheeck small white bumps/WHITE PATCH ON THE PLATE OF MY MOUTH/
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Diagnosis and Treatment of Oral Leukoplakia

Wednesday, March 3rd, 2010

leukoplakia

Diagnosis

The white color of the leukoplakia is caused by a thickening of the layers of prickle cell keratinocytes or taking a white color when wet. Histologically, the majority of benign biopsies show hyperkeratosis, acanthosis and chronic inflammation. Approximately 20% of cases show a dysp

lasia that can range from mild to moderate. The tendency of lesions to become malignant can be investigated by various methods, including the determination of mitotic index, apoptotic and Ki67 and the expression of proto-oncogenes p53 and Bax and Bcl2

From a clinical standpoint, the diagnosis of leukoplakia is done by excluding other diseases that produce similar lesions.

Candidiasis: the first step in establishing the differential diagnosis of a white lesion in oral mucosa is whether it can break off (as in pseudomembranous candidiasis) or not.

Lichen: if the lesion is located in the mucous membranes of both cheeks differential diagnosis must be established with lichen planus and lupus erythematosus. The final diagnosis was determined by biopsy and immunofluorescence

Injury: the most common are mucosal chewed and focal keratosis.dentistry

Inherited as white lesions white sponge nevus: family history and histologogía can distinguish from leukoplakia

Treatment

Elimination of risk factors causing discontinuation of snuff up to 50% regression of leukoplakia in which snuff is the etiological factor.

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Incoming search terms for the article:

leukoplakia and reactive arthritis/leukoplakia regression/oral focal keratosis/woods lamp and oral leukoplakia/
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What Is Leukoplakia?

Monday, March 1st, 2010

Oral leukoplakia

dentistLeukoplakia is defined as a stain or white plaque, which appears on the tongue or mucous membranes of the mouth, which can not be scraped or detached and can not be attributed clinically or pathologically by any other disease. The significance of this lesion is its relatively high incidence and it is considered a precancerous lesion at 5% of cases, progresses to squamous cell carcinoma.

From a clinical standpoint, leukoplakia is classified as:

* Homogeneous leukoplakia, the most frequent, with little risk of malignant transformation
* Nodular or mottled, very rare, with a relatively high risk of degeneration to cancer
* Erythroleukoplakia, similar to the homogeneous leukoplakia but surrounded by red lesions

Some authors include as verrucous leukoplakia fourth group, proliferative and aggressive very rare, with increased risk of malignant transformation and are often seen as an advanced stage of homogeneous leukoplakia.

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