Hyperpigmentation is an overload of pigment in the skin, usually becomes visible on the face especially cheeks, nose and perioral region. It is also common in pregnant women, where the spots usually appear on the nipples, scars, stretch marks and in the abdominal area. These spots usually are yellowish brown to dark brown.

Apart from pregnancy, other causes of hyperpigmentation, including gynecological disorders, thyroid function and / or adrenal deficient and sun exposure.

The spots are intensified in the summer due to solar radiation is more intense. Drinking to excess sun can have several consequences, including: premature skin aging, immune system depression, occasionally severe burns and skin cancer.
One of the main causes of these stains are nasty ultraviolet rays from the sun and not have adequate sun protection can trigger the process. Other triggers are related to kidney problems and hormonal imbalances, for example following a pregnancy.

Exposure to the sun without sunscreen can produce large amounts of melanin which is what controls the pigmentation of the skin and as a result may appear the spots. When the problem is mild can be treated with creams or bleaching products that reduce melanogenesis (the process responsible for the creation of melanin), which causes the skin discoloration.

One of the best known whitening hydroquinone that is not without side effects and therefore be prescribed only by prescription. Other bleaches containing pea extract, sorbic acid or lemon extract derived from that usually produce good results by reducing blemishes.

Other tips to maintain healthy skin is cleaned with exfoliants or moisturizers on a regular basis and if natural products, the better. We should also avoid sun exposure during extremely harmful, we also take care to drink plenty of water and care for food (balanced diet, plenty of fruit, vegetables and some vitamin supplements)

Influenza is a disease of old people and other animals (birds, pigs …) and that causes epidemics every year, mainly during the winter months. This disease is different types of viruses that also can be combined between them to form a new flu.

Influenza is highly contagious and is spread easily from person to person through respiratory droplets or secretions that ousted the talking, coughing or sneezing.
Symptoms

Recognizing this condition is easy, because it behaves like the flu either. Consider ratifying it if it suddenly had:

* 1. At least one of these four symptoms:
Fever over 38 º C.
or malaise.
or headache.
or muscle aches.
* 2. And besides, at least one of these three symptoms:
Cough.
or sore throat.
o Difficulty breathing.

Occasionally they can appear nausea and vomiting.
Every so often the virus can change into many different types from those that existed in previous years and compared to that, being new, we have no defense for what is spread far more people than usual. Everyone can get.

Throughout history there have been several pandemics, such as the 1918 Spanish flu. The pandemic caused many deaths were not available because of health systems with which we now have and I had a high virulence.

The current pandemic is caused by a new subtype of influenza A (H1N1), and so far has a mild, although this could change over the months.

Seasonal Flu

Seasonal influenza is presented each year during the winter months. Every year people die because of complications caused by it and therefore it is recommended to get vaccinated persons are more likely to suffer complications.

This year also is expected to be seasonal flu. Many people are already protected by defenses have generated in previous years. However, as every year, has developed a vaccination campaign targeting the usual population groups.

Although the seasonal flu vaccine does not protect against the new influenza virus A (H1N1), unanimously recommended that all persons belonging to the usual risk groups are vaccinated, as they do every year.

It is possible that both flu could overlap in time.
The antiviral and vaccine

Drugs that are being used to treat influenza in the manner established clinical protocols are called antivirals.

Two drugs: oseltamivir (trade name Tamiflu) and zanamivir (with the brand name Relenza). None of these drugs cure. Act by reducing the duration of the disease in about a day and the possibility of complications. Therefore, currently only indicated in people with influenza requiring hospitalization or those which, until now, have a higher chance of complications.

In these individuals is considered that, before the diagnosis of influenza, the benefit of the administration of these drugs outweighs the risk of potential side effects have any medicine.

Like all medicines, Tamiflu and Relenza, have side effects and contraindications, therefore, must be a physician to decide when to take them. Expert Groups reassess continuously, protocols and indications for treatment.

The pandemic influenza vaccine (H1N1) is available from 16 November to the population groups identified as priorities by the Inter-Territorial Council of the National Health System (CISNS) to receive pandemic vaccine, which initially are:

Pregnant women
* Socio-health workers
All workers or health facilities, both primary and hospital care, public and private.
Staff employed in homes and elderly care centers and chronically ill persons who have contact with vulnerable people.

* People working in essential public services
or forces and state security bodies, with national unit, regional or local level.
or Fire.
or civil protection services.
or people working on mobile services health emergencies.
or workers from other prisons and detention centers by court order.

* People older than 6 months of age, to present a special medical condition, have an increased risk of complications from infection with the pandemic virus (H1N1) 2009. These clinical conditions are:
or chronic cardiovascular disease (excluding hypertension).
or chronic respiratory diseases (including broncho-pulmonary dysplasia, cystic fibrosis and moderate-severe persistent asthma).
or diabetes mellitus type I and type II with medical therapy.
or moderate-severe renal insufficiency.
or Hemoglobinopathies and moderate-severe anemia.
o Lack of spleen.
or advanced chronic liver disease.
or severe neuromuscular diseases.
or immunocompromised patients (including those caused by HIV infection, or drugs, or transplant recipients).
or morbid obesity (body mass index less than 40).
o Persons under 18 years receiving prolonged treatment with aspirin, for the chance of developing Reye syndrome.

In our country, vaccination is voluntary, even in groups where it is recommended as a priority, as has happened in every vaccination campaigns have been carried out.

The vaccine does not always prevent the disease, but gives it some protection by reducing the risk of complications.

If in doubt, consult your doctor.
Actions Ministry of Health and Social Policy and Health Councils of the Autonomous Communities

Ministry of Health and Social Policy and Health Councils of the Autonomous Communities.

For years it has a plan for preparedness and response to pandemic influenza. The Expert Committees and Subcommittees are working, for months, in the preparation and updating of specific protocols, monitoring of the pandemic and in proposing and implementing measures, both clinical and social, to help manage the pandemic.

Spain has a reserve of 15 million antiviral treatment to be provided to all who need it, once valued by health professionals.

The Government, by unanimous agreement of all the Autonomous Communities, has commissioned new virus vaccines against influenza A (H1N1) to 18 million people. The calculation was made taking into account the population most likely to suffer complications and those working in essential services to the community in these circumstances.

Our National Health System is ready and adapting every day to face this situation and all administrations are coordinated.

How is it diagnosed?

The diagnosis of AS is complicated by the lack of an evaluation or standardized diagnostic program. In fact, because there are various assessment tools in use today, each with a different approach, the same child may receive different diagnoses depending on the useful assessment used by the doctor.

To complicate matters further, some doctors believe that AS is not a separate and distinct disorder. Instead, they call high-functioning autism and see it at the end of the autism spectrum with mild symptoms that differ only in degree, from classic autism. Some clinicians use two diagnoses, AS or high functioning autism so interchangeable. This makes the collection of data on the incidence of AS, as some children will be diagnosed with high functioning autism instead of AS, and vice versa.

Most doctors rely on the presence of a core group of behaviors to alert them about the possibility of a diagnosis of AS. These are:

* Abnormal eye contact

* Withdrawal

* Do not turn around when called by name

* Do not use gestures to point or show

* Lack of interactive game

* Lack of interest in others

Some of these behaviors may be apparent in the first months of life the child, or appear later. Before 3 years of age must be present problems in at least one of the areas of communication and socialization or repetitive behavior and restricted.

The diagnosis of AS is a two stage process. The first stage begins with the assessment of development during a review of “healthy child” with the family physician or pediatrician. The second stage is a comprehensive assessment team to consider or rule out AS. This team usually includes a psychologist, neurologist, psychiatrist, speech therapist, and other professionals with expertise in diagnosing children with AS.

A comprehensive evaluation includes neurological and genetic assessment, with detailed tests of cognitive and language to establish IQ and psychomotor function assessed, strengths and weaknesses verbal and nonverbal learning style, and independent living skills. An assessment of strengths and weaknesses of communication includes assessing nonverbal forms of communication (gaze and gestures), the use of non-literal language (metaphors, irony, absurdities and humor); patterns of inflection, stress and volume modulation; pragmatic (taking shifts and sensitivity to verbal suggestions) and content, clarity and coherence of conversation. The doctor will see the results of the tests and combined with the child’s developmental history and current symptoms for diagnosis.

Are there treatments?

The ideal treatment for AS coordinates therapies that address the three core symptoms of the disorder: poor communication skills, obsessive or repetitive routines and physical clumsiness. There is no ideal treatment package for all children with AS, but most professionals agree that the earlier one intervenes, the better.

An effective treatment program builds on children’s interests, offers a predictable, teaches tasks as a series of simple steps, actively engages the child’s attention in highly structured activities and provides regular reinforcement of behavior. This type of program usually includes:

* Social skills training, a form of group therapy that teaches children with AS the skills they need to interact more successfully with other children

* Cognitive behavioral therapy, a type of therapy of “talk” that can help children more anxious or explosives to better manage their emotions and reduce obsessive interests and repetitive routines

* Medication, for coexisting conditions like depression and anxiety

* Occupational or physical therapy for children with sensory integration problems or poor motor coordination

* Specialized therapy and speech / language, to help children who have problems with the pragmatics of language, exchange of normal conversation

* Training and support for parents to teach them behavior techniques for use at home

Is it better for children with AS? What happens when they reach adulthood?

With effective treatment, children with AS can learn to cope with their disabilities, but may still find social situations and personal relationships require great effort. Many adults with AS are capable of working successfully in work under, although they still need encouragement and moral support to maintain independent living.

What research is being done?

The National Institute of Neurological Disorders and Stroke is one of the main sponsors of federal biomedical research on disorders of the brain and nervous system. The NINDS conducts research in its laboratories at the National Institutes of Health (NIH) in Bethesda, Maryland, and provides grants to support research in universities and other facilities. Many of the NIH Institutes, including NINDS, are sponsoring research to understand what causes AS and how it can be treated effectively.

A study using functional magnetic resonance imaging (fMRI) to show how abnormalities in particular areas of the brain cause changes in brain function that produce symptoms of AS and other autistic disorders. Another large-scale study is comparing psychiatric and neuropsychological assessments of children with possible diagnosis of AS or high functioning autism to those of their parents and siblings to see if symptom patterns linking AS and high functioning autism profiles specific neuropsychological.

NINDS is also supporting an international study of long-range linking researchers to collect and analyze DNA samples from children with AS and high functioning autism, like their families to identify genes associated and how they interact. Called Autism Genome Project is a consortium of scientists from universities, academic centers and institutions around the world that functions as a reservoir of genetic data for researchers to discover the basis of AS and other autistic disorders.

Because there are so many different forms of autistic disorders, understanding the genetic basis of each opens up opportunities for more accurate diagnosis and treatment. Knowing the genetic profile of a particular disorder can mean the early identification of those at risk, and early intervention when it is likely that therapies and treatments are most successful.

Asperger syndrome is a severe developmental disorder that has as main feature the difficulty in social interaction and finding interest, followed by a limited and unusual behavior. Many come to confuse this condition with autism without mental RETAS calling high functioning autism, but is now somewhat difficult to establish differences between the two since they have similar symptoms.
People with this syndrome tend to hide, since they possess good oral communication, an IQ within the normal and even often higher than normal, and a solitary lifestyle, as this is common behavior of human beings, it becomes very difficult to know who has this syndrome. But the shortcomings can be seen in new situations or social situations that require other behavior which is not usual.
We emphasize that people with Asperger syndrome manifested maladaptive behaviors in various environments, and this is because these individuals have a poor understanding of social phenomena and therefore astonished when facing new experiences.

Asperger’s observations, published in German, is not widely known until 1981, when the English doctor Lorna Wing published a series of case studies of children who showed similar symptoms, which called syndrome of “Asperger’s. Wing’s writings were widely published and popularized. AS became a distinct disease diagnosed in 1992, when it was included in the tenth edition of the diagnostic manual published by the World Health Organization, International Classification of Diseases (ICD-10), and in 1994 was added to the Diagnostic Manual and Statistical of Mental Disorders IV (DSM-IV) diagnostic reference book of the American Psychiatric Association.

What are some common signs or symptoms?

The most distinguishing symptom of AS is the child’s obsessive interest in a single object or topic to the exclusion of any other. Some children with AS have become experts on vacuum cleaners, makes and models of cars, even objects as peculiar as fryers. Children with AS want to know everything about their topic of interest and their conversations with others will be about little else. Their expertise, high level of vocabulary and formal speech patterns make them seem like little professors.

Children with AS gather large amounts of factual information about your favorite topic and talk about it incessantly, but the conversation may seem like a collection of facts and figures at random, without point or conclusion.

Their speech may be marked by the lack of rhythm, a peculiar inflection, or a monotone. Often children with AS lack the ability to modulate the volume of his voice to emulate the surrounding area. For example, should be reminded to talk softly every time they come to a library or a theater.

Unlike the great retreat from the world which is characteristic of autism, children with AS are isolated because of their poor social skills and few interests. In fact, maybe reach out to other people, but make normal conversation impossible due to inappropriate or eccentric behavior, or trying to speak only of interest only.

Children with AS usually have a history of developmental delays in motor skills such as pedaling a bicycle, catch a ball or climb a outdoor play equipment. They are often clumsy and have poor coordination with a march which may seem forced or jumping.

Many children with AS are highly active in early childhood and then develop anxiety or depression as young adults. Other conditions that often coexist with AS are ADHD, tic disorders (Tourette’s syndrome), depression, anxiety disorders and obsessive-compulsive disorder.

What causes AS? Is it genetic?

Current research points to brain abnormalities as the cause of AS. Using advanced brain imaging technique, scientists have revealed structural and functional differences in specific regions of the brains of normal children compared to children with AS. These defects are most probably caused by abnormal migration of embryonic cells during fetal development that affects brain structure and the “wiring” and then going to affect the neural circuits that control thought and behavior.

For example, one study found reduced brain activity in the frontal lobe of children with AS when asked to respond to tasks that required the use of discretion. Another study found differences in activity when the children were asked to respond to facial expressions. A separate study investigating brain function in adults with AS revealed abnormal levels of specific proteins that correlate with obsessive and repetitive behaviors.

Scientists have always known that there must be a genetic component to AS and other autistic disorders due to inherited. Additional evidence was observed for the linkage between inherited genetic mutations and AS in the higher incidence of family members who have behavioral symptoms similar to AS but more limited. For example, they had mild difficulties with social interaction, language and reading.

However, it has never identified a specific gene for AS. However, recent research indicates that there is probably a common set of genes whose changes or deletions make a person vulnerable to develop AS. This combination of genetic variations or deletions will determine the severity and symptoms of individuals with AS.

LVO usually does not require treatment or cause serious symptoms. However, treatment may be an option for those concerned with the appearance of the LVO in the language, or who have disseminated lesions and experience discomfort or taste changes due to the plates.

The usual method for treating viral LVO is taken orally (by mouth). Usually, these medicines are taken for one to two weeks or until the plates of the LVO disappear.
Other options include tretinoin (Retin A) and podophyllin resin, two drugs that can be applied directly to the plates of LVO. Usually tretinoin is applied two or three times a day until the plates are gone. Podophyllin it operates a health care provider once or twice during a period of two to three weeks. Other options, especially if the plates are small, is that a healthcare provider applies liquid nitrogen (cryotherapy) to the affected area, or surgery to remove the plates.

Can you prevent the LVO?

There is no sure way to prevent the plates of the LVO. However, the best way to prevent it can be to maintain healthy immune system. This means keeping viral load low and high T cells, using anti-HIV treatment, and adopting a healthy lifestyle.

Is there any treatment for developing LVO?

Currently, there are drugs in development to treat LVO as an opportunistic infection is not serious, and that there are medications to treat it. If you are interested in participating in any clinical trials with new therapies for the treatment or prevention of LVO, there is an interactive site aimed for finding clinical trials is AIDSinfo.nih.gov a site run by the National Institutes of Health United States (National Institutes of Health). These pages are “specialists in health information, with whom you can communicate by calling toll free 1-800-HIV-0440 (1-800-448-0440).

Oral hairy leukoplakia (LVO) is one or more white patches that can appear in the mouth. Normally, these plaques appear along the border of the tongue, but sometimes develop on the top or bottom of the tongue or along the inside of the cheek. If you look closely, these plaques can appear hairy or contain a number of small folds or bumps.

The LVO may look like thrush (oral candidiasis), another common problem characterized by white patches that develops in the mouths of people with HIV. However, if canker scraped gently using a toothbrush, usually are removed, the LVO not.

The LVO is one of the first opportunistic infections that occur in people with HIV. May occur with any T-cell count While there are people with HIV with more than 500 T cells that developed LVO, is more common in those with T-cell counts under 200. It is noteworthy that the LVO may affect those with healthy immune systems, even those who are not infected with HIV.

It is considered a benign disease, since it rarely causes serious physical problems and cause no serious complications.

The Epstein-Barr virus (EBV) is causing the LVO. Most people worldwide are infected with EBV. But only some, including those with a weakened immune system, develop the disease.

Over 25% of HIV positive people LVO develops sometime during the course of infection. It is more common among HIV positive men and smokers.

What are the symptoms of LVO?

The classic symptom of the LVO are looking fluffy white patches (hairy) and bumps that develop on the edge of the tongue. Plates may also appear at the top or bottom of the tongue, or along the inside of the cheek. It is also possible that these plates do not exhibit villi and contain no visible bumps or creases.

In general, LVO causes no other symptoms. Therefore, many people may not know they have LVO unless you consider the tongue or inside the mouth to find the plates, which usually do not cause discomfort or affect the taste of food or liquids. In some cases, LVO may cause mild pain, taste changes and sharpen sensitivity to food temperatures.

How is it diagnosed the LVO?

Often, health professionals can diagnose by simply looking LVO white plates. To find out if LVO plates or thrush (candidiasis) may be a simple test of scaling. With the help of a trowel or brush teeth gently scraping the plate. If the board is clear with white scaling, it is probably thrush, not LVO.

To be sure if the white patch is LVO, your health care provider can send a sample to a lab for analysis. The lab will look for the Epstein-Barr virus (EBV) to confirm the diagnosis of LVO.

Diagnosis

The white color of the leukoplakia is caused by a thickening of the layers of prickle cell keratinocytes or taking a white color when wet. Histologically, the majority of benign biopsies show hyperkeratosis, acanthosis and chronic inflammation. Approximately 20% of cases show a dysp

lasia that can range from mild to moderate. The tendency of lesions to become malignant can be investigated by various methods, including the determination of mitotic index, apoptotic and Ki67 and the expression of proto-oncogenes p53 and Bax and Bcl2

From a clinical standpoint, the diagnosis of leukoplakia is done by excluding other diseases that produce similar lesions.

Candidiasis: the first step in establishing the differential diagnosis of a white lesion in oral mucosa is whether it can break off (as in pseudomembranous candidiasis) or not.

Lichen: if the lesion is located in the mucous membranes of both cheeks differential diagnosis must be established with lichen planus and lupus erythematosus. The final diagnosis was determined by biopsy and immunofluorescence

Injury: the most common are mucosal chewed and focal keratosis.

Inherited as white lesions white sponge nevus: family history and histologogía can distinguish from leukoplakia

Treatment

Elimination of risk factors causing discontinuation of snuff up to 50% regression of leukoplakia in which snuff is the etiological factor.

Retinoic acid: retinoic acid, vitamin A and other synthetic retinoids are a treatment option, but only get remissions in 40-60% of cases. Tretinoin has been administered in doses of 1 mg / kg / day orally for 2 to 3 months or, alternatively, topically in solution or 0.05% cream. However, to discontinue treatment usually occurs a recurrence of injuries.

Photodynamic Therapy: Photodynamic therapy has been introduced recently in the treatment of cancers and oral leukoplakia. This treatment is based on observation of Sutro in 1933 in breast cancers that cancerous tissue illuminated with Wood’s lamp emitting a red light, whereas normal tissues did green. Although the biochemical basis of this phenomenon is only partially known, has shown that neoplastic or dysplastic tissues showed a reduction of fluorescence especially in the green region of the spectrum, while only the red light is affected. Photodynamic therapy is based on the administration of a photosensitizing agent that causes selective irradiation is to tissues where it accumulates this. The light produces reactive oxygen species and other free radicals that destroy DNA, structural proteins and phospholipids.

Have published several studies on photodynamic therapy in the treatment of leukoplakia using sensitizing agent d-aminolevulinic acid (a precursor of heme) applied locally at the site of injury and an argon laser at 635 nm. In some studies, the percentage of referrals reached 80%. Although the number of clinical studies of photodynamic therapy is limited, the results suggest that this treatment may be superior to others, and is also well tolerated (only 30% of patients experience pain during treatment)

Ablative surgery: removal of oral leukoplakia can be accomplished by surgical excision. The carbon dioxide laser or Nd: YAG lasers have the advantage of speed, restricted hemorrhages, removal of the lesion at constant depth, low morbidity and reduced scar contraction. For the elimination of proliferative verrucous leukoplakia surgical excision is preferred because in this type of leukoplakia is higher recurrence when using the laser. Cryosurgery also has also been used, although it is used less and less due to a possible worsening of the dysplasia and the impossibility of obtaining samples for biopsies.

After eliminating patients should be monitored at intervals of 3 to 6 months. The recurrence ranges between 20-35% after surgical excision and between 9-22% after laser excision. Very often, the recurrent lesions were found in areas adjacent to the primary lesion. In all cases, only considered that healing has been complete if not observed any recurrence within 3 years after treatment. (bhakti)

Leukoplakia is defined as a stain or white plaque, which appears on the tongue or mucous membranes of the mouth, which can not be scraped or detached and can not be attributed clinically or pathologically by any other disease. The significance of this lesion is its relatively high incidence and it is considered a precancerous lesion at 5% of cases, progresses to squamous cell carcinoma.

From a clinical standpoint, leukoplakia is classified as:

* Homogeneous leukoplakia, the most frequent, with little risk of malignant transformation
* Nodular or mottled, very rare, with a relatively high risk of degeneration to cancer
* Erythroleukoplakia, similar to the homogeneous leukoplakia but surrounded by red lesions

Some authors include as verrucous leukoplakia fourth group, proliferative and aggressive very rare, with increased risk of malignant transformation and are often seen as an advanced stage of homogeneous leukoplakia.

Homogeneous leukoplakia is a lesion uniformly white, firm, not very thick and smooth or rough. Nodular leukoplakia is a raised lesion with inhomogeneous edges presented with small rounded white and red elevations of the mucosa that give a granular appearance. It is also called speckled leukoplakia. The erythroleukoplakia is characterized by being surrounded by erythematous areas, sometimes with erosions. Finally, the proliferative verrucous leukoplakia, first described in 1984 is an exophytic lesion, persistent, warty and clinically aggressive and resistant to treatment.

Etiopathology

Of leukoplakias can be classified as induced by external factors and idiopathic. The snuff is one of major causes of this disease, with snuff leukoplakias by between 6 and 10 times more frequent than those resulting from other causes.

Other factors that have been viewed as inducing factors of leukoplakia are excessive alcohol consumption, poor diet with vitamin or mineral deficiencies, bad habits of compulsive (nibble parts of the mouth), poor oral hygiene and ill-fitting dentures or the existence of sharp angles of the crowns and fillings. The Viodent (a mouthwash based on an active principle of Sanguinaria canadensis) has been implicated in the development of oral leukoplakia.
Many authors consider some infectious diseases (infections with Candida, syphilis, infections with human papillomavirus (HPV) as etiological factors of leukoplakia (bhakti)

* Urinary incontinence.
* Bladder dysfunction.
* Intestinal dysfunction.
* Dizziness.
* Dysphagia.
* Fatigue.
* Difficulty in walking.
* Headache.
* Loss of hearing.
* Itching.
* Numbness.
* Optic neuritis.
* General pain and a tendency to chronicity.
* Tremor.
* Difficulty speaking.
* Depression.

Diet or recommended nutrients

For multiple sclerosis is advised purifying diet, green leafy vegetables for their richness in potassium, non-acidic fruits, whole grains and fish.
The omega-3 fatty acids, Omega 6, antioxidants like vitamin E, selenium, zinc and sulfur amino acids such as L-acetylcysteine for their effect to slow nerve degeneration and to act as anti-inflammatory.

Vitamin D and calcium as people with multiple sclerosis tend to have more chances of osteoporosis.

Vitamin B12 and studies have shown the link between vitamin D deficiency and multiple sclerosis.

Natural treatments for multiple sclerosis

The treatment is based on improving the symptoms during the acute outbreak and try to slow the emergence of new shoots.

In allopathic medicine is usually given steroids and medication to increase defenses, are often given intravenously in some cases, not always needed and can be administered in day centers for the treatment of multiple sclerosis.

It’s always alleviate all symptoms for a better quality of life of patients suffering from multiple sclerosis.

Oligoterapia for multiple sclerosis

Copper-gold-silver is very effective in energy deficit.
Manganese will add when they have joint pain.

Phytotherapy and Medicinal Plants

If Lavender is recommended neuralgia: the Sativa, or Wild Oats for its contribution remineralizing and nervous system strengthener.
To reinforce the mood in multiple sclerosis, you should make teas with Hipérico Damiana, equally.
You can also take Valerian as sleep inducer and enhancer of Passionflower Valerian.

Aromatherapy for multiple sclerosis

Essential oils of Lavender and Lemon Balm (a few drops on the pillow cases of insomnia)
Oils of Marjoram, Lavender and Ylang Ylang in the bath to relax tension and muscle.

Other natural therapies

Also important are Yoga and meditation to eliminate stress that the disease behaves.
Passive exercise at home without effort, but constant, is a good preventive therapy for people with multiple sclerosis.
A great will acupuncture to slow the worsening crisis. If the principle is significantly reduced symptoms and increases energy and brings the channels involved in multiple sclerosis.

In all cases we recommend checking with your doctor, therapist or other qualified health provider. The information in this article is for information only.

Credit to: Manoli Duart

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Definition of Multiple Sclerosis

Multiple sclerosis is a disease of slow onset with a tendency to chronicity. Affected the immune system, renal system, nervous system, the joint system and the excretory system.

It is a disease of the restrictive nature which also has to take into account the negative thoughts and feelings generated by multiple sclerosis.

The most important factor in the emergence of multiple sclerosis is the loss of myelin is composed of protein and fat.

Myelin facilitates the conduction of nerve impulses produced in our brains. Damage to the myelin gives us multiple sclerosis. Everyone suffers from different symptoms and may suffer from mild to severe symptoms.

Causes and etiology of Multiple Sclerosis

The exact cause of the onset of multiple sclerosis is not known exactly, but it is believed that the destruction of myelin is due to the body’s own immune response that defends itself and does not recognize the tissue as its own, destroying this case the myelin sheath of the nervous system.

Recent studies indicate a higher manifestation of multiple sclerosis in some areas than in others, may be due to the high content of toxic substances like mercury, lead, aluminum as they are considered one of the causes of the emergence of multiple sclerosis.

The onset of MS is slow, mostly diagnosed in adulthood and presents with mild difficulty then disappear for a while.
We can find nutritional deficiencies such as decompensation of the fatty acids in our body.

There are more number of cases with multiple sclerosis in Western industrialized countries and the large amount of processed foods, red meats, low intake of vegetables, etc..

Clinical Manifestations of Multiple Sclerosis

A person with MS may have one or several symptoms at once. Successive crises may develop new symptoms appear.
We must also bear in mind that not everyone gets the same symptoms but vary from one person to another.
They tend to divide the symptoms of multiple sclerosis in three categories:

* Primary Symptoms
Are those that result due to demyelination:
Weakness, numbness, tremors, vision loss, pain, paralysis, bladder dysfunction, bowel dysfunction.
Some of these symptoms can be alleviated or solved with proper treatment.

* Secondary Symptoms
Derivatives are the complications of the primary symptoms, such as:
Bladder dysfunction, urinary weakness, decreased bone density.

* Tertiary symptoms
Complications are social, vocational and psychological symptoms resulting from primary and secondary.
The tension that causes multiple sclerosis affects social relations to suffer from a disease of limitations on short and long term.
Depression is common in these patients.

Credit to: Manoli Duart